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108 itemsUnited States Army Gas School Course Notes
- 1918
Notebook consists of handwritten notes taken by Francis E. Scott at the U.S. Army Gas School at Camp Kendrick, New Jersey, on the use of gas in warfare. The notes include teaching aids and a circular letter from…
- Author Scott, Francis E.
- Subject Science--Study and teaching, World War (1914-1918), Chemical warfare--Safety measures, Military education, Intrenchments, Fries, Amos A. (Amos Alfred), 1873-1963, Gas masks--Maintenance and repair, United States. Army. Chemical Warfare Service, Scott, Francis E., Chemical agents (Munitions), Chemical warfare--Equipment and supplies, Great Britain. Army. Royal Engineers, Chemical warfare--Safety measures--Equipment and supplies, Phosgene, Gases, Asphyxiating and poisonous--War use--Safety measures, United States. Army, Chlorine, Gases, Asphyxiating and poisonous--War use, Mustard gas, Trenches, Chemical warfare, Artillery, Chemical weapons, Chloropicrin
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3 itemsPlate II (Fig. 1): Trachea and lungs of dog dying 12 hours after gassing.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Chlorine poisoning.
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3 itemsPlate XXXVII (Fig. 6): Pneumonia confined to one lobe and appearing as large yellow abscesses on the surface in dog dying 5 days after exposure.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Poisoning from organic arsenic compounds.
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Plate XLI (Fig. 2): Hemorrhages in wall of damaged bronchus 6 days after chlorpicrin gassing. (Fig. 3) Higher magnification of hemorrhage in wall of bronchus shown in Fig. 2.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Hemorrhages in residual pulmonary lesions from respiratory irritating gases.
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Plate XL (Fig. 1): Hemorrhages in lung of dog killed 10 days after phosgene gassing.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Hemorrhages in residual pulmonary lesions from respiratory irritating gases.
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Plate XXXVIII (Figs. 8, 9): Congestion and hemorrhage about the bronchial tree standing out sharply against relatively normal lung tissue.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Poisoning from organic arsenic compounds.
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Plate XXXIX (Fig. 10): Hemorrhage into peribronchial sheath without extension to alveolar tissue; also perivascular edema.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Poisoning from organic arsenic compounds.
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Plate XXXVI (Fig. 4): Pneumonia arising diffusely throughout tissue full of edema.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Poisoning from organic arsenic compounds.
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Plate XXXV (Fig. 3): Great congestion of capillaries in alveolar walls and edema fluid in alveoli.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Poisoning from organic arsenic compounds.
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Plate XXXIV (Fig. 2): Trachea with edematous membrane in dog dying 19 hours after exposure to phenyldichlorarsine.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Poisoning from organic arsenic compounds.
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Plate XXXIII (Fig. 1): Trachea and lungs from dog dying 1 day after exposure to ethyldichlorarsine.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Poisoning from organic arsenic compounds.
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Plate XXXI (Fig. 3): Spleen, kidney, and section of aorta of dog surviving 49 hours after inhalation of arsine.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Arsine poisoning.
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3 itemsPlate XXV (Fig. 19): Necrotizing membrane of epiglottis and larynx and extensive pneumonia 5 days after a 30-minute exposure to mustard gas.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Mustard poisoning.
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Plate XXXII (Fig. 4): Kidney of dog. Death occurred in 5 days after exposure to arsine. (Fig. 5) Section of liver of dog dying 2 days after exposure to a lethal dose of arsine.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Arsine poisoning.
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Plate XXX (Fig. 2): Section of kidney of dog succumbing after 10 hours to a lethal dose of arsine.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Arsine poisoning.
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Plate XXVIII (Fig. 3): Lungs of dog surviving 7 hours after a lethal dose of cyanogen bromide.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Cyanogen (chloride and bromide) poisoning.
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Plate XXIX (Fig. 1): Spleen, kidney, and wall of aorta from a dog dying 8 hours after inhalation of arsine.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Arsine poisoning.
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Plate XXVII (Fig. 1): Lungs of dog, surviving 5 hours, gassed with a high concentration of cyanogen chloride.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Cyanogen (chloride and bromide) poisoning.
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Plate XXVI (Fig. 22): Eye of dog dying 4 days and 21 hours after exposure to mustard gas. (Fig. 23) Eye of animal dying 7 days after exposure to mustard gas. (Fig. 24) Water color drawing of trachea showing constriction below larynx; 1 month after exposure.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Mustard poisoning.
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Plate XXIV (Fig. 18): Pneumonia with early abscess formation in dog dying 5 days after exposure to mustard gas.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Mustard poisoning.
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Fig. 16: Dilatation of small bronchus with epithelial necrosis; small masses of bacteria (to left) with leucocytic infiltration; death 72 hours after exposure. Fig. 17: Small group of cocci in lung 24 hours after exposure.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Mustard poisoning.
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Fig. 20: Chronic inflammatory cells infiltrating submucosa of trachea; the epithelium is lacking. Fig. 21: Same trachea as shown in Fig. 20.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Mustard poisoning.
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Fig. 9: Red cells ingested by macrophages. Fig. 10: Bronchus showing extent of damage.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Preliminary studies in intratracheal therapy.
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Fig. 14: Early pneumonic exudate of polymorphonuclear leucocytes. Fig. 15: Hemorrhage into lung, alveoli full of red cells.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Mustard poisoning.
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Fig. 5: Congestion in alveolar walls. Fig. 6: Bronchus showing extent of damage.
- Part of Collected Studies on the Pathology of War Gas Poisoning
- 1920
Preliminary studies in intratracheal therapy.